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免疫異常による神経疾患(神経-免疫-内分泌,<特集>東京女子医科大学学会第61回総会)
http://hdl.handle.net/10470/23375
http://hdl.handle.net/10470/233759ffcd7e3-c467-4cc4-a5f3-28e3350a0bd4
名前 / ファイル | ライセンス | アクション |
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Item type | 学術雑誌論文 / Journal Article(1) | |||||
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公開日 | 2010-08-10 | |||||
タイトル | ||||||
タイトル | 免疫異常による神経疾患(神経-免疫-内分泌,<特集>東京女子医科大学学会第61回総会) | |||||
言語 | ||||||
言語 | jpn | |||||
資源タイプ | ||||||
資源タイプ識別子 | http://purl.org/coar/resource_type/c_6501 | |||||
資源タイプ | journal article | |||||
別タイトル | ||||||
その他のタイトル | Immune Abnormality in Neurological Diseases(Neuro-Immuno-Endocrinology) | |||||
著者名 |
太田, 宏平
× 太田, 宏平 |
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著者別名 | ||||||
姓名 | OTA, Kohei | |||||
出版者 | ||||||
出版者 | 東京女子医科大学学会 | |||||
受付日付 | ||||||
日付 | 2010-08-10 | |||||
日付タイプ | Created | |||||
ISSN | ||||||
収録物識別子タイプ | ISSN | |||||
収録物識別子 | 0040-9022 | |||||
NCID | ||||||
収録物識別子タイプ | NCID | |||||
収録物識別子 | AN00161368 | |||||
書誌情報 |
東京女子医科大学雑誌 巻 66, 号 4, p. 203-214, 発行日 1996-04-25 |
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著者版フラグ | ||||||
出版タイプ | VoR | |||||
出版タイプResource | http://purl.org/coar/version/c_970fb48d4fbd8a85 | |||||
抄録 | ||||||
内容記述タイプ | Abstract | |||||
内容記述 | Multiple sclerosis (MS) is thought to be an autoimmune disease mediated by T cells respond to central nervous myelin antigens including myelin basic protein (MBP) and proteolipid protein because of their encephalitogenic potentials in experimental autoimmune encephalomyelitis, animal model of MS. We report a higher frequency of T cell lines reactive to MBP between residues 84-102 in patients with MS in both Caucasians and Japanese, so it is likely that MBP reactive T cells for this region have an important role in the etiology of patients with MS. Guillain-Barre syndrome (GBS) in which anti-glycolipid antibodies were proved in recently years is considered as another typical neuroimmunological disease however it is still examined whether anti-neuronal antibody (ANA) is a primary cause of demyelination in GBS. In addition to GBS, other specific ANAs were detected in paraneoplastic neurological syndrome such as LambertEaton myasthenic syndrome, paraneoplastic cerebellar degeneration and paraneoplastic sensory neuropathy. And investigations of such ANAs in the various neuroimmunological disorders are becoming useful diagnostic tools. | |||||
著者所属 | ||||||
東京女子医科大学脳神経センター神経内科 |